Fish Oil
Daily intake of essential fatty acids could help reduce the need for painkillers in patients suffering from rheumatoid arthritis (RA), according to a new study.
Published in the journal Rheumatology, the free-access study found that a daily 10g intake of cod liver oil was associated with a 30 per cent reduction in the use of non-steroidal anti-inflammatory drugs (NSAIDs).
The findings could present a new market opportunity for essential fatty acids, especially in light of mounting concerns about the adverse effects of NSAID use. Some of these drugs, which are among the most frequently prescribed medications worldwide, have been linked with gastrointestinal toxicity, increased blood pressure, and increased risk of cardiovascular disease.
A combination of fish-oil supplements and exercise may help improve cardiovascular health more than either one alone, an Australian study suggests.
The study, published in this month’s issue of the American Journal of Clinical Nutrition, included 75 overweight men and women, ages 25 to 65, who had at least one of the following cardiovascular risk factors — mild hypertension, elevated total cholesterol, and elevated plasma triacylglycerols.
Br J Nutr. 2008 Mar 17;:1-9
Long-chain n-3 polyunsaturated fatty acid from fish oil modulates
aortic nitric oxide and tocopherol status in the rat.
In spite of their high oxidisability, long-chain n-3 PUFA protect
against CVD. Dietary fatty acids modulate the fatty acid composition
of lipoproteins involved in atherosclerosis. We thought that if long-
chain n-3 PUFA were able to increase NO production by the aorta, then
by its antioxidant activity the NO will prevent lipid peroxidation.
However, the beneficial effect of NO in vivo on VLDL+LDL oxidation
would only be possible if NO could diffuse to their lipidic core. Rats
were fed maize oil- or fish oil as menhaden oil- (MO) rich diets for 8
weeks, to study the effects of MO on aortic NO production, NO
diffusion into VLDL+LDL, the extent of oxidation in native VLDL+LDL
and their oxidisability ex vivo. Aortic NO production and its alpha-
tocopherol content were increased and n-3 PUFA were incorporated into
the VLDL+LDL. In spite of the higher peroxidisability and the low
alpha-tocopherol in native VLDL+LDL from rats fed MO, native VLDL+LDL
from the two groups shared similar electrophoretic patterns,
conjugated dienes, thiobarbituric acid-reactive substances, total
antioxidant capacity, and NO diffusibility on VLDL+LDL, indicative of
an in vivo protection against oxidation. However, these results do not
correlate with the ex vivo oxidisability of VLDL+LDL, as NO is
lacking. Thus, the in vivo beneficial effects can be explained by
increased alpha-tocopherol in aorta and by a compensatory effect of NO
on VLDL+LDL against the low alpha-tocopherol levels, which may
contribute to the anti-atherogenic properties of fish oil.
PMID: 18341754
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